Metabolic alterations related to T2D are very well characterised by epidemiological and research based studies. The pathogenesis and progression of T2D is ascribed to 4 mechanisms; elevated Sophisticated glycation conclusion products (AGE) development, increased polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. Until https://www.directivepublications.org/journal-of-diabetology-research/