Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. Conversely, our capability to rescue SERT purpose in KI midbrain synaptoneurosomes from the inhibition of FAK suggests elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by improved pFAK localization in 5-HT synapses. Our knowledge https://ermak531pbm3.glifeblog.com/profile